Complement activity is regulated in C3 glomerulopathy by IgG–factor H fusion proteins with and without properdin targeting domains - ScienceDirect

Bi-allelic pathogenic variations in DNAJB11 cause Ivemark II syndrome, a renal-hepatic-pancreatic dysplasia - Kidney International

C3 glomerulopathies (C3G) are caused by dysregulation of the

Frontiers Modeling C3 glomerulopathies: C3 convertase regulation on an extracellular matrix surface

Structure of complement fragment C3b–factor H and implications for host protection by complement regulators

PDF) Complement-mediated kidney diseases

The role of the alternative pathway of complement activation in glomerular diseases

PDF) Complement-mediated kidney diseases

C3 glomerulopathy and atypical hemolytic uremic syndrome: an updated review of the literature on alternative complement pathway disorders

C3d-Targeted factor H inhibits tissue complement in disease models and reduces glomerular injury without affecting circulating complement - ScienceDirect

Proposed model for complement activation in hemodialysis (HD). The

PDF) Complement activation and regulation in rheumatic disease

PDF) Complement therapy in atypical haemolytic uraemic syndrome (aHUS)